neutralizing antibodies against cytokine Search Results


antibodies against il 17a  (ATCC)
95
ATCC antibodies against il 17a
Antibodies Against Il 17a, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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primary antibodies against il-17a  (Huabio Inc)
90
Huabio Inc primary antibodies against il-17a
Primary Antibodies Against Il 17a, supplied by Huabio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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il-17a neutralizing antibody  (Amgen)
90
Amgen il-17a neutralizing antibody
Il 17a Neutralizing Antibody, supplied by Amgen, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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rabbit polyclonal anti il-10  (Abcam)
98
Abcam rabbit polyclonal anti il-10
Rabbit Polyclonal Anti Il 10, supplied by Abcam, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti–il-10 ab  (Becton Dickinson)
90
Becton Dickinson anti–il-10 ab
Anti–Il 10 Ab, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti-il-10 neutralizing ab jes5-16e3  (Thermo Fisher)
90
Thermo Fisher anti-il-10 neutralizing ab jes5-16e3
Anti Il 10 Neutralizing Ab Jes5 16e3, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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p mapk14  (Cell Signaling Technology Inc)
99
Cell Signaling Technology Inc p mapk14
Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, <t>p-MAPK14),</t> cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit <t>MAPK14</t> or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments
P Mapk14, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/p mapk14/product/Cell Signaling Technology Inc
Average 99 stars, based on 1 article reviews
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antibodies against il-17a e93203  (EnoGene Inc)
90
EnoGene Inc antibodies against il-17a e93203
Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, <t>p-MAPK14),</t> cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit <t>MAPK14</t> or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments
Antibodies Against Il 17a E93203, supplied by EnoGene Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/antibodies against il-17a e93203/product/EnoGene Inc
Average 90 stars, based on 1 article reviews
antibodies against il-17a e93203 - by Bioz Stars, 2026-03
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anti-il-17a neutralizing abs  (Novartis)
90
Novartis anti-il-17a neutralizing abs
Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, <t>p-MAPK14),</t> cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit <t>MAPK14</t> or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments
Anti Il 17a Neutralizing Abs, supplied by Novartis, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
anti-il-17a neutralizing abs - by Bioz Stars, 2026-03
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neutralizing anti-il-10  (Becton Dickinson)
90
Becton Dickinson neutralizing anti-il-10
Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, <t>p-MAPK14),</t> cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit <t>MAPK14</t> or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments
Neutralizing Anti Il 10, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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neutralizing anti-il-10 (23738)  (R&D Systems)
90
R&D Systems neutralizing anti-il-10 (23738)
Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, <t>p-MAPK14),</t> cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit <t>MAPK14</t> or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments
Neutralizing Anti Il 10 (23738), supplied by R&D Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse anti bovine il 10 antibody  (Bio-Rad)
93
Bio-Rad mouse anti bovine il 10 antibody
Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, <t>p-MAPK14),</t> cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit <t>MAPK14</t> or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments
Mouse Anti Bovine Il 10 Antibody, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, p-MAPK14), cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit MAPK14 or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments

Journal: BMC complementary and alternative medicine

Article Title: Transcriptomic and proteomic insight into the effects of a defined European mistletoe extract in Ewing sarcoma cells reveals cellular stress responses.

doi: 10.1186/s12906-017-1715-2

Figure Lengend Snippet: Fig. 4 ViscumTT induces cellular stress responses. a TC-71 and MHH-ES-1 cells were treated with increasing concentrations of viscumTT (ML-I 1– 40 ng/mL + OA 10–60 μg/mL), viscum (ML-I 1–40 ng/mL) or TT (10–60 μg/mL) for 24 h. Activation of stress-mediated MAPK signalling (p-MAPK8, p-MAPK14), cellular stress/unfolded protein response (EIF2AK3, HSPA5) and autophagy (LC3BI/II) were assessed using western blotting. β-actin was used as loading control, and images shown are representative for three independent experiments. Mistletoe lectin (ML) and oleanolic acid (OA) concentrations were used as a measure of viscum and TT active agent extract concentration, respectively. b TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the presence of DMSO (inhibitor solvent control), 10 μM SB203580 or 5 μM SP600125 to inhibit MAPK14 or MAPK8 activation, respectively. Inhibition of MAPK activation was not detected within the used inhibitor concentrations in whole-cell extracts using western blotting. β-actin was used as loading control, and images are representative for results from three independent experiments. c TC-71 cells were treated with viscumTT, viscum or TT (~ IC50 concentration) for 24 h in the absence or presence of the TLR4 inhibitor, LPS-RS (0.1 μg/mL), or the antioxidant, N-acetylcysteine (NAC, 5 mM). Apoptosis was flow cytometrically assessed following annexin V/propidium iodide staining. Bars show the percentage of apoptosis inhibition is shown in bars (±SEM) from three independent experiments

Article Snippet: Primary antibodies were directed against p-MAPK14 (Thr180/Tyr182, #9211 Cell Signaling Technology, Danvers, MA, USA), LC3B (#2775 Cell Signaling Technology), EIF2AK3 (#3192, Cell Signaling Technology), pMAPK8 (sc-6254, Santa Cruz biotechnology, Dallas, TX, USA), HSPA5 (#G8918, Sigma-Aldrich) and ß-actin conjugated directly to peroxidase (#A3854, Sigma-Aldrich).

Techniques: Activation Assay, Western Blot, Control, Concentration Assay, Solvent, Inhibition, Staining